Deep Brain Stimulation (DBS) for Movement Disorders

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Deep Brain Stimulation (DBS): Young Parkinson's Patient Finds Relief with Surgery

Deep Brain Stimulation: What to Expect

The DBS treatment is an ongoing therapy and not a single surgical event.

Learn about the four phases of treatment including assessment, surgery, initial programming and long-term maintenance.

Frequently Asked Questions About DBS

Find answers to commonly asked questions related to Deep Brain Stimulation for those with Parkinson's:

  • Does DBS work for everyone?
  • Is DBS safe and effective?
  • Why choose the RWJ DBS team?

The DBS Team

We have developed a comprehensive program that supports patients for the duration of the treatment.

Meet the multisciplinary team of DBS physicians with specialties including neurosurgery, neurology, neurophysiology, psychiatry and speech and hearing.

 

How can we help you?How Can We Help You?

Request more information or an appointment. A member of our team will respond to you promptly.

Contact Us Online or call us at
1-888-MD-RWJUH.

 

 

 

Deep Brain Stimulation for Parkinson's DiseaseWhat is Deep Brain Stimulation (DBS)?

Deep Brain Stimulation (DBS) is an adjunctive surgical treatment that is FDA approved for the movement disorders of Dystonia, Essential Tremor (ET) and Parkinson's Disease (PD). DBS does not replace medical therapy, but frequently allows reductions in medication and related side effects.

DBS is the surgical implantation of precisely placed stimulating leads into deep brain targets located within the basal ganglia: either the Globus Pallidus (GPi) or the subthalamic nucleus (STN). The stimulating electrodes are connected to small cables passed underneath the skin and plugged into an Internal Pulse Generator (IPG), which is also implanted underneath the skin on the upper torso.

The IPG is programmed to send a continuous stream of small, brief pulses to the tips of the stimulating leads.

In PD, dopamine loss in the substantia nigra allows the STN and the GPi to disengage from normal control and become dysfunctional. In generalized DYT1 and focal dystonia, GABAergic dysfunction results in a loss of inhibiton in cortex and the basal ganglia. ET is a clinically defined syndrome with many etiologies, or underlying causes, a number of which respond to DBS.

DBS modulates the basal ganglia circuit, by limiting the influence of the STN and GPi, thereby re-balancing the circuit towards more normal functioning.

Click here to learn about what is involved with DBS treatment.